1 edition of Clinical and metabolic aspects of fructose found in the catalog.
Clinical and metabolic aspects of fructose
|Series||Acta medica Scandinavica. Supplementum -- 542|
|Contributions||Huttunen, Jussi K., Nikkilä, E|
|The Physical Object|
|Pagination||244 p. :|
|Number of Pages||244|
Book Description. The first edition of this innovative book brought a new perspective to the metabolic and therapeutic aspects of amino acids in clinical nutrition. Since its publication, a number of very important advances have been made . Fructose and metabolic diseases: New findings, new questions Luc Tappy M.D.a,b, Kim A. Lê Ph.D.a, Christel Tran M.D.a, Nicolas Paquot M.D.c What are the mechanisms responsible for fructose-induced metabolic alterations? There is overwhelming evidence that, in rodents, a high-sucrose or a high-fructose diet will lead to the File Size: KB.
Essential reading for candidates for the MRCPath examination and similar postgraduate examinations in clinical biochemistry. The book gives an overview of the acquisition of data, as well as concentrating on clinical aspects of the subject, giving detailed coverage of all conditions where clinical biochemistry is used in diagnosis and management. The mechanisms underlying fructose-induced metabolic disturbances are unclear but are beginning to be unraveled. In contrast to metabolism of glucose, the breakdown of fructose leads to the generation of metabolites that stimulate hepatic de novo lipogenesis (DNL) and increased levels of both fasting and postprandial triglycerides.
Diabetes and Metabolic Syndrome: Clinical Research and Reviews is the official journal of DiabetesIndia.. Diabetes and Metabolic Syndrome: Clinical Research and Reviews aims to reach out to healthcare professionals, diabetes educators and other stakeholders, providing them with a global platform to submit their research on diabetes care. The journal publishes research covering various aspects. The metabolic and health effects of both nutritive and non-nutritive sweeteners are controversial, and subjects of intense scientific debate. These potential effects span not only important scientific questions, but are also of great interest to media, the public and potentially even regulatory bodies. Fructose, High Fructose Corn Syrup, Sucrose and Health serves as .
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Essential reading for candidates for the MRCPath examination and similar postgraduate examinations in clinical biochemistry. The book gives an overview of the acquisition of data, as well as concentrating on clinical aspects of the subject, giving detailed coverage of all conditions where clinical biochemistry is used in diagnosis and by: The clinical and metabolic aspects of fructose have been discussed at several symposia 3–6.
Keywords Lactic Acid Uric Acid Metabolic Acidosis Carbohydrate Metabolism Acute EpisodeCited by: 1. Acta Med Scand Suppl. ; Clinical and metabolic aspects of fructose. Concluding remarks. Froesch ER. PMID: [PubMed - indexed for MEDLINE]Cited by: 3.
Sugar-sweetened vs. diet soft drinks and serum uric acid levels. • Hyperuricemia is considered the precursor of gout • Soft drinks contain large amounts of fructose, which is the only carbohydrate known to increase uric acid levels • 14, adults from the Third NHANES.
Choi JW, et al. Arthritis & Rheumatism Three recent clinical studies, which investigated the effects of consuming relevant doses of sucrose or high fructose corn syrup along with ad libitum diets, provide evidence that consumption of these sugars increase risk factors for cardiovascular disease (CVD) and metabolic by: This is a novel pathway linked to excess fructose intake and its metabolic relevance remains to be clarified [ 15 ].
In summary, fructose seems to influence multiple metabolic pathways in the liver that results in enhanced lipogenesis, generation of uric acid, ER stress, and by: 1.
Among various factors, such as an unhealthy diet or a sedentarity lifestyle, excessive fructose consumption is known to favor nonalcoholic fatty liver disease (NAFLD), as fructose is both a substrate and an inducer of hepatic de novo lipogenesis.
The present review presents some well‐established mechanisms and new clues to better understand the pathophysiology of fructose Cited by: Clinical Biochemistry: Metabolic and Clinical Aspects.
Book • 3rd Edition • Edited by: this leading textbook primarily focuses on clinical aspects of the subject, giving detailed coverage of all conditions where clinical biochemistry is used in diagnosis and management – including nutritional disorders, diabetes, inherited.
Fructose is a monosaccharide that is present in high concentrations in fruit and honey and is a constituent of sucrose and sorbitol. Fructose metabolism disorders are one of the many carbohydrate metabolism disorders. Fructose is seen as uniquely contributing to the pandemics of obesity and its cardiometabolic complications.
Much of the evidence for this view derives from the unique biochemical, metabolic, and. The clinical and metabolic aspects of fructose have been discussed at several symposia3–6.
Purchase Clinical Biochemistry:Metabolic and Clinical Aspects - 3rd Edition. Print Book & E-Book. ISBN The metabolic syndrome is a common syndrome affecting about 20 % of the adult population in Europe, and probably the prevalence is of the same magnitude in other industrialised countries worldwide.
It is mainly caused by western lifestyle resulting in abdominal obesity, but also a genetic predisposition plays a role. Dietary fructose, in contrast, may produce a lesser postprandial rise in plasma glucose than other common carbohydrates.
There is considerable controversy aboutthe effects of dietary sucrose and fructose on serum lipids, and their effects on other metabolic events, such as the nonenzymatic glycosylation of proteins, are by: Through a unique presentation of clinical photographs and accompanying histologic photomicrographs, Clinical and Pathological Aspects of Skin Diseases in Endocrine, Metabolic, Nutritional and Deposition Disease provides the practicing surgical pathologist, dermatologist, endocrinologist, internal medicine physician and dermatopathologist with a single volume Format: Paperback.
The principal form has been in the form of high-fructose corn syrup found in soft drinks and processed food. The effect of excessive fructose consumption on human health is only beginning to be understood. Fructose has been confirmed to induce several obesity-related complications associated with the metabolic syndrome.
Ahima has a clinical interest in obesity, diabetes and related metabolic diseases. He is an attending endocrinologist at the Hospital of the University of Pennsylvania, director of Obesity Unit of the Institute for Diabetes, Obesity and Metabolism, and director of the Penn Diabetes Research Center Mouse Phenotyping Core.
Free Online Library: Fructose: a key factor in the development of metabolic syndrome and hypertension.(Report) by "Journal of Nutrition and Metabolism"; Health, general B cells Physiological aspects Glucose metabolism Hypertension Development and progression Diet therapy Insulin resistance. Dietary Fat, Salt and Sugar: A clinical perspective of the social catastrophe.
Influences of Food Ingredients on Enterohepatic Circulation of Bile Acids. Anemia: Influence of Dietary Fat, Sugar and Salt on Hemoglobin and Blood Health. Food Behavior, Food Addiction and Metabolic Syndrome.
Diabetes mellitus and the metabolic syndrome are becoming leading causes of death in the world. Identifying the etiology of diabetes is key to prevention. Despite the similarity in their structures, fructose and glucose are metabolized in different ways.
Uric acid, a byproduct of uncontrolled fructose metabolism is known risk factor for by:. Clinical Biochemistry: Metabolic and Clinical Aspects William J. Marshall, Márta Lapsley, Andrew Day, Ruth Ayling Now fully revised and updated, Clinical Biochemistry, third edition is essential reading for specialty trainees, particularly those preparing for .Purpose of review.
Fructose is seen as uniquely contributing to the pandemics of obesity and its cardiometabolic complications.
Much of the evidence for this view derives from the unique biochemical, metabolic, and endocrine responses that differentiate fructose from glucose. To understand whether these proposed mechanisms result in clinically meaningful modification of cardiovascular risk .Chronic fructose consumption can affect metabolic gene expression programs that further affect fructose disposition.
These mechanisms will be described in greater detail below. Although the liver metabolizes the majority of ingested fructose, the intestine itself can metabolize up to 30% of an oral fructose load (56, 57).Cited by: